Peritoneal fibrosis is mouse strain dependent.
نویسندگان
چکیده
an analysis of the Netherlands Cooperative Study on the Adequacy of Dialysis (NECOSAD)-2. J Am Soc Nephrol 2004; 15: 1061–70 13. Daugirdas JT. Simplified equations for monitoring Kt/V, PCRn, eKt/V and ePCRn. Adv Ren Replace Ther 1995; 2: 295–304 14. Tattersall JE, DeTakats D, Chamney P et al. The post-hemodialysis rebound: predicting and quantifying its effect on Kt/V. Kidney Int 1996; 50: 2094–2102 15. Leypoldt J, Jaber BL, Zimmerman DL. Calculation of standard Kt/V (stdKt/V) with corrections for postdialysis urea rebound. Hemodial Int 2003; 7: 80 16. F Gotch. Kinetic modeling in hemodialysis. In: Nissenson AR, Fine RN, Gentile DE (eds). Clinical Dialysis. 3rd edn. Appleton AR and Lange RN, Norwalk, CT. 1996: 156–189 17. JT Daugirdas, Greene T, Chertow GM et al. Can rescaling dose of dialysis to body surface area in the HEMO study explain the different responses to dose in women versus men? Clin J Am Soc Nephrol 2010; 5: 1628–1636 18. Ramirez SP, Kapke A, Port FK et al. Dialysis dose scaled to body surface area and size-adjusted, sex-specific patient mortality. Clin J Am Soc Nephrol 2012; 7: 1977–87 19. A Einstein. Über die von der molekularkinetischen Theorie der Wärme geforderte Bewegung von in ruhenden Flüssigkeiten suspendierten Teilchen. Annalen der Physik 1905; 322: 549–560 20. Eloot S, Van Biesen W, Vanholder R. A sad but forgotten truth: the story of slow-moving solutes in fast hemodialysis. Semin Dial 2012; 25: 505–9 21. Schneditz D, Yang Y, Christopoulos G et al. Rate of creatinine equilibration in whole blood. Hemodial Int 2009; 13: 215–21 22. Eloot S, Schneditz D, Vanholder R. What can the dialysis physician learn from kinetic modeling beyond Kt/V urea? Nephrol Dial Transplant 2012; 27: 4021–4029 23. Babb AL, Popovich RP, Farrell PC et al. The effects of erythrocyte mass transfer rates on solute clearance measurements during haemodialysis. Proc Eur Dial Transplant Assoc 1972; 9: 303–321 24. van Olden RW, Krediet RT, Struijk DG et al. Measurement of residual renal function in patients treated with continuous ambulatory peritoneal dialysis. J Am Soc Nephrol 1996; 7: 745–50
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عنوان ژورنال:
- Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association
دوره 28 8 شماره
صفحات -
تاریخ انتشار 2013